Journal of Bacteriology, March 2008, p. 1838-1842, Vol. 190, No. 5
0021-9193/08/$08.00+0 doi:10.1128/JB.01551-07
Copyright © 2008, American Society for Microbiology. All Rights Reserved.
The AraC-Like Transcriptional Regulator DhbR Is Required for Maximum Expression of the 2,3-Dihydroxybenzoic Acid Biosynthesis Genes in Brucella abortus 2308 in Response to Iron Deprivation
Eric S. Anderson,
James T. Paulley, and
R. Martin Roop II*
Department of Microbiology and Immunology, East Carolina University School of Medicine, Greenville, North Carolina 27834
Received 26 September 2007/
Accepted 11 December 2007
Phenotypic evaluation of isogenic mutants derived from Brucella abortus 2308 indicates that the AlcR homolog DhbR (2,3-dihydroxybenzoic acid [2,3-DHBA] biosynthesis regulator) modulates the expression of the genes involved in 2,3-DHBA production, employing 2,3-DHBA or brucebactin as a coinducer.
* Corresponding author. Mailing address: Department of Microbiology and Immunology, East Carolina University School of Medicine, 600 Moye Boulevard, Greenville, NC 27834. Phone: (252) 744-1357. Fax: (252) 744-3535. E-mail: roopr{at}ecu.edu
Published ahead of print on 21 December 2007.
Journal of Bacteriology, March 2008, p. 1838-1842, Vol. 190, No. 5
0021-9193/08/$08.00+0 doi:10.1128/JB.01551-07
Copyright © 2008, American Society for Microbiology. All Rights Reserved.
Copyright © 2008 by the American Society for Microbiology. All rights reserved.