Journal of Bacteriology, June 2008, p. 4367-4371, Vol. 190, No. 12
0021-9193/08/$08.00+0 doi:10.1128/JB.01898-07
Copyright © 2008, American Society for Microbiology. All Rights Reserved.

School of Dentistry, Meharry Medical College, Nashville, Tennessee,1 Department of Oral Biology, University of Florida, Gainesville, Florida2
Received 4 December 2007/ Accepted 27 March 2008
Streptococcus cristatus ArcA inhibits production of a major adhesin, FimA, in Porphyromonas gingivalis, a primary periodontal pathogen. In this study, we demonstrate the differential expression of arcA in two streptococcal species. The expression level of arcA in streptococci appears to be controlled by both cis and trans elements.
Published ahead of print on 11 April 2008.
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