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Journal of Bacteriology, March 2008, p. 1838-1842, Vol. 190, No. 5
0021-9193/08/$08.00+0     doi:10.1128/JB.01551-07
Copyright © 2008, American Society for Microbiology. All Rights Reserved.

The AraC-Like Transcriptional Regulator DhbR Is Required for Maximum Expression of the 2,3-Dihydroxybenzoic Acid Biosynthesis Genes in Brucella abortus 2308 in Response to Iron Deprivation{triangledown}

Eric S. Anderson, James T. Paulley, and R. Martin Roop II*

Department of Microbiology and Immunology, East Carolina University School of Medicine, Greenville, North Carolina 27834

Received 26 September 2007/ Accepted 11 December 2007

Phenotypic evaluation of isogenic mutants derived from Brucella abortus 2308 indicates that the AlcR homolog DhbR (2,3-dihydroxybenzoic acid [2,3-DHBA] biosynthesis regulator) modulates the expression of the genes involved in 2,3-DHBA production, employing 2,3-DHBA or brucebactin as a coinducer.

* Corresponding author. Mailing address: Department of Microbiology and Immunology, East Carolina University School of Medicine, 600 Moye Boulevard, Greenville, NC 27834. Phone: (252) 744-1357. Fax: (252) 744-3535. E-mail: roopr{at}ecu.edu

{triangledown} Published ahead of print on 21 December 2007.

Journal of Bacteriology, March 2008, p. 1838-1842, Vol. 190, No. 5
0021-9193/08/$08.00+0     doi:10.1128/JB.01551-07
Copyright © 2008, American Society for Microbiology. All Rights Reserved.





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