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JB Accepts, published online ahead of print on 21 December 2007
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J. Bacteriol. doi:10.1128/JB.01551-07
Copyright (c) 2007, American Society for Microbiology and/or the Listed Authors/Institutions. All Rights Reserved.

The AraC-like transcriptional regulator DhbR is required for maximum expression of the 2, 3-dihydroxybenzoic acid (2,3-DHBA) biosynthesis genes in Brucella abortus 2308 in response to iron deprivation

Eric S. Anderson, James T. Paulley, and R. Martin Roop II*

Department of Microbiology and Immunology, East Carolina University School of Medicine, Greenville, North Carolina 27834

* To whom correspondence should be addressed. Email: roopr{at}ecu.edu.


  Abstract

Phenotypic evaluation of isogenic mutants derived from Brucella abortus 2308 indicates that the AlcR homolog DhbR (2,3-DHBA biosynthesis regulator) modulates expression of the genes involved in 2,3-dihydroxybenzoic acid production employing 2,3-DHBA or brucebactin as a co-inducer.




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