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resistance

  • <em>Pseudomonas</em> Can Survive Tailocin Killing via Persistence-Like and Heterogenous Resistance Mechanisms
    Research Article
    Pseudomonas Can Survive Tailocin Killing via Persistence-Like and Heterogenous Resistance Mechanisms

    Bacteriocins are bacterially produced protein toxins that are proposed as antibiotic alternatives. However, a deeper understanding of the responses of target bacteria to bacteriocin exposure is lacking. Here, we show that target cells of Pseudomonas syringae survive lethal bacteriocin exposure through both physiological persistence and genetic resistance mechanisms....

    Prem P. Kandel, David A. Baltrus, Kevin L. Hockett
  • Fis Contributes to Resistance of <span class="named-content genus-species" id="named-content-1">Pseudomonas aeruginosa</span> to Ciprofloxacin by Regulating Pyocin Synthesis
    Research Article
    Fis Contributes to Resistance of Pseudomonas aeruginosa to Ciprofloxacin by Regulating Pyocin Synthesis

    Pseudomonas aeruginosa is an important opportunistic pathogenic bacterium that causes various acute and chronic infections in human, especially in patients with compromised immunity, cystic fibrosis (CF), and/or severe burn wounds. About 60% of cystic fibrosis patients have a chronic respiratory infection caused by...

    Yuqing Long, Weixin Fu, Su Wang, Xuan Deng, Yongxin Jin, Fang Bai, Zhihui Cheng, Weihui Wu
  • Open Access
    Metallochaperones Are Needed for <span class="named-content genus-species" id="named-content-1">Mycobacterium tuberculosis</span> and <span class="named-content genus-species" id="named-content-2">Escherichia coli</span> Nicotinamidase-Pyrazinamidase Activity
    Research Article
    Metallochaperones Are Needed for Mycobacterium tuberculosis and Escherichia coli Nicotinamidase-Pyrazinamidase Activity

    Tuberculosis is an infectious disease caused by the bacterium Mycobacterium tuberculosis and remains one of the major causes of disease and death worldwide. Pyrazinamide is a key drug used in the treatment of tuberculosis, yet its mechanism of action is not fully understood, and testing strains of M....

    Patricia Sheen, Anuntxi Monsalve, Jhanina Campos, Rodolfo Huerta, Ricardo Antiparra, Héctor Arteaga, Patricia Duran, Carlos Bueno, Daniela E. Kirwan, Robert H. Gilman, Mirko Zimic
  • “It Takes a Village”: Mechanisms Underlying Antimicrobial Recalcitrance of Polymicrobial Biofilms
    Minireview
    “It Takes a Village”: Mechanisms Underlying Antimicrobial Recalcitrance of Polymicrobial Biofilms

    Chronic infections are frequently caused by polymicrobial biofilms. Importantly, these infections are often difficult to treat effectively in part due to the recalcitrance of biofilms to antimicrobial therapy. Emerging evidence suggests that polymicrobial interactions can lead to dramatic and unexpected changes in the ability of antibiotics to eradicate biofilms and often result in decreased antimicrobial efficacy in vitro....

    Giulia Orazi, George A. O’Toole
  • Entry Exclusion of Conjugative Plasmids of the IncA, IncC, and Related Untyped Incompatibility Groups
    Research Article
    Entry Exclusion of Conjugative Plasmids of the IncA, IncC, and Related Untyped Incompatibility Groups

    IncA and IncC conjugative plasmids drive antibiotic resistance dissemination among several pathogenic species of Gammaproteobacteria due to the diversity of drug resistance genes that they carry and their ability to mobilize antibiotic resistance-conferring genomic islands such as SGI1 of Salmonella enterica. While historically grouped as “IncA/C,” IncA and...

    Malika Humbert, Kévin T. Huguet, Frédéric Coulombe, Vincent Burrus
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